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<!DOCTYPE ArticleSet PUBLIC "-//NLM//DTD PubMed 2.7//EN" "https://dtd.nlm.nih.gov/ncbi/pubmed/in/PubMed.dtd">
<ArticleSet>
<Article>
<Journal>
				<PublisherName>Iranian Heart Association</PublisherName>
				<JournalTitle>Iranian Heart Journal</JournalTitle>
				<Issn></Issn>
				<Volume>16</Volume>
				<Issue>3</Issue>
				<PubDate PubStatus="epublish">
					<Year>2015</Year>
					<Month>09</Month>
					<Day>01</Day>
				</PubDate>
			</Journal>
<ArticleTitle>Left Ventricular Clot due to Wild-Type Homozygous Factor V Leiden Mutation: A Case Report</ArticleTitle>
<VernacularTitle></VernacularTitle>
			<FirstPage>57</FirstPage>
			<LastPage>59</LastPage>
			<ELocationID EIdType="pii">83164</ELocationID>
			
			
			<Language>EN</Language>
<AuthorList>
<Author>
					<FirstName>Farahnaz</FirstName>
					<LastName>Nikdoust</LastName>
<Affiliation>Department of Cardiology, Shariati Hospital, Tehran University of Medical Sciences, Tehran,I.R.Iran.</Affiliation>

</Author>
<Author>
					<FirstName>Mahdieh</FirstName>
					<LastName>Emami</LastName>
<Affiliation>Department of Cardiology, Shariati Hospital, Tehran University of Medical Sciences, Tehran,I.R.Iran.</Affiliation>

</Author>
<Author>
					<FirstName>Seyed Abdolhussein</FirstName>
					<LastName>Tabatabaei</LastName>
<Affiliation>Department of Cardiology, Shariati Hospital, Tehran University of Medical Sciences, Tehran,I.R.Iran.</Affiliation>

</Author>
</AuthorList>
				<PublicationType>Journal Article</PublicationType>
			<History>
				<PubDate PubStatus="received">
					<Year>2019</Year>
					<Month>02</Month>
					<Day>28</Day>
				</PubDate>
			</History>
		<Abstract>Factor V Leiden (FVL) mutation has been identified as a frequent risk factor for life-threatening venous thromboembolic events. We describe a 39-year-old man admitted with sudden-onset blurred vision and mild lower-limb paresthesia with a positive activated protein C resistance test. The polymerase chain reaction confirmed wild-type homozygous FVL mutation. Transthoracic echocardiography revealed a highly elongated mobile mass attached to the akinetic cardiac apex, which extended to the left ventricular outflow tract. The thrombosis was removed by thrombectomy through aortotomy. The patient was in good clinical condition atthe last follow-up. (Iranian Heart Journal 2015;16(3):57-59)</Abstract>
		<ObjectList>
			<Object Type="keyword">
			<Param Name="value">Factor V Leiden</Param>
			</Object>
			<Object Type="keyword">
			<Param Name="value">Mutation</Param>
			</Object>
			<Object Type="keyword">
			<Param Name="value">Thrombosis</Param>
			</Object>
		</ObjectList>
<ArchiveCopySource DocType="pdf">https://journal.iha.org.ir/article_83164_a456e3aa3c4eabc5db513f2eba293f7c.pdf</ArchiveCopySource>
</Article>
</ArticleSet>
